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Modulating Autophagy as a Treatment for Lysosomal Storage Diseases
Researchers at NIAMS have developed a technology for treatment of lysosomal storage diseases by inhibition of autophagy. Pompe disease is an example of a genetic lysosomal storage disease caused by a reduction or absence of acid alpha-glucosidase (GAA). Patients with Pompe disease have a lysosomal buildup of glycogen in cardiac and skeletal muscle...
Published: 8/14/2024
|
Inventor(s):
Cynthia Schreiner
,
Paul (Estate of) Plotz
,
Shoichi Takikita
,
Tao Xie
,
Rebecca Baum
,
Nina Raben
Keywords(s):
Acid
,
A-glucosidase
,
Autophagy
,
B
,
Clearance
,
Complete
,
DEFICIENCY
,
Disabling
,
Disease
,
Enzyme
,
ERT
,
GB1XXX
,
GBXXXX
,
Genetic
,
GLYCOGEN
,
GXXXXX
,
IBXXXX
,
Inactivation
,
IXXXXX
,
Lysosomal
,
Model
,
Mouse
,
MUSCLE
,
Near
,
Patent Category - Biotechnology
,
Permits
,
Pompe
,
REPLACEMENT
,
SKELETAL
,
Storage
,
STORED
,
THERAPY
,
UA1XXX
Category(s):
Collaboration Sought > Collaboration
,
Application > Therapeutics
,
TherapeuticArea > Rare / Neglected Diseases
,
Application > Diagnostics
,
Application > Research Materials